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Stands to reason. We know that morbidly obese patients face 50x the risk of malignancies of the ideal weight population, but these are mostly hormonal-associated (ovarian, breast, prostate), along with colon. Is it the sugar environment along with the relatively hormone-laden fat stores?
maybe the measured growth of cells in a high sugar environemnt versus a high fat environment may give clue.. Thinking along the lines that sugar is easy for the cell to "eat" maybe even that unatural sugars enhance the growth of the mutated cells ???
We know that morbidly obese patients face 50x the risk of malignancies of the ideal weight population, but these are mostly hormonal-associated (ovarian, breast, prostate), along with colon. Is it the sugar environment along with the relatively hormone-laden fat stores
Exactly. It's possible that the impact of simple sugars on triglyceride and prostaglandin metabolism is at the crux of this. Triacylglycerols or triglycerides are supposed to function as depot or storage lipids. Fatty acyl-CoA and glycerols are used to manufacture trigs. (Lehninger's text of biochem does a better job of explaining the theory I'm embracing here - I'm looking at my copy, the second edition, as well as an old edition of Goodman & Gilman).
Excess intake of simple sugars potentially contributes to the production of more acetyl CoA, which is the biosynthetic precursor of prostaglandins, steroid hormones and cholesterol, than we need. Our bodies are set up to store this excess as trigs. In this way excess triacylglycerols produced in response to sugar excess, function as a overabundant reserve of essential fatty acids. This could result in excessive production of prostaglandins, cholesterol & hormones.
If these stored fatty acids produce inflammatory prostaglandins, they would contribute to CVD, CAD (by enzymatic processes that aspirin inhibits) and, if these stored fatty acids scoot down another metabolic pathway, their excess could result in relative hormone excess or a relative prostaglandin-mediated overstimulation of reproductive glands. Even though the impact of sugar on prostaglandin & hormone production would be relative or mild, if it is persistent, this could explain a considerable proportion of diet/lifestyle associated disease.
Thinking about who controls the sugar market..
Booze,soda, chocolate ice cream junk food the beet growers \corn growers \food industry..
Even if you can prove that simple sugars are the silent killer and the REAL cancer.. What hope would you have ..
I am not a fan of any diet plan per se but years back I used to drive truck and during the evening I listened to Dr Atkins on the radio and his comments made sense also his logic was simple...\
Even starting back the the origins of man stuff any sugar form was total random and a very short lived event..
I think in the long term tho the ultimate solution is to realise that food is nothing more than gas in the gas tank of the car ..
When was the last time you had the familiy over to pump gas..
I know I am opening all kind of doors here for Kurst.
But once folks realise that food is not a social event but rather a bodily function.. Then the food intake would be relative to needs of the body rather than the wants of the eyes and nostrils..
be great to just eat some pills and that the food for the day ..What a time saver then at night hook to to your nutrio drip.. So wake up sustained and fed..
Hmm when was the last time you had a bunch of of people over to perform bodily functions..
Oh thats right that happens in DC its called the goverment..
Feeding infants and toddlers study: What foods are infants and toddlers eating?
Mary Kay Fox, MEda
Susan Pac, MS, RDb
Barbara Devaney, PhDc
Linda Jankowski, MSc
Objective: To describe the food consumption patterns of US infants and toddlers, 4 to 24 months of age.
Design: Descriptive analysis of data collected in the 2002 Feeding Infants and Toddlers study based on telephone interviews and 24-hour dietary recalls.
Subjects: A national random sample of 3,022 infants and toddlers age 4 to 24 months.
Statistical analyses performed: The percentage of infants and toddlers consuming foods from specific food groups was estimated for six age groups, using a single 24-hour recall.
Results: Infants as young as 7 months of age showed food patterns that have been observed in older children and adults. From 18% to 33% of infants and toddlers between ages 7 and 24 months consumed no discrete servings of vegetables, and 23% to 33% consumed no fruits. French fries were one of the three most common vegetables consumed by infants 9 to 11 months of age. By 15 to 18 months, french fries were the most common vegetable. Almost half (46%) of 7- to 8-month-olds consumed some type of dessert, sweet, or sweetened beverage, and this percentage increased as age increased. By 19 to 24 months, 62% of toddlers consumed a baked dessert, 20% consumed candy, and 44% consumed a sweetened beverage.